rabbit anti-gfp (chip) (Thermo Fisher)
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rabbit anti-gfp (chip)
Rabbit Anti Gfp (Chip), supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+anti-gfp+(chip)/rabbit+anti+gfp/pm40239647-597-182-46
Average 90 stars, based on 1 article reviews
Rabbit Anti Gfp (Chip), supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+anti-gfp+(chip)/rabbit+anti+gfp/pm40239647-597-182-46
Average 90 stars, based on 1 article reviews
rabbit anti-gfp (chip) - by Bioz Stars,
2026-10
90/100 stars
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other:Article Title: Global regulation via modulation of ribosome pausing by the ABC-F protein EttA Article Snippet: Blinding was not relevant for this study, since the outcomes of the experiments are not dependent of the judgment of the researcher but depend of control reactions. re p o rtfo lio | rep o rtin g su m m ary A pril2023 Materials & experimental systems n/a Involved in the study Antibodies Eukaryotic cell lines Palaeontology and archaeology Animals and other organisms Clinical data Dual use research of concern Plants Methods n/a Involved in the study ChIP-seq Flow cytometry MRI-based neuroimaging Antibodies Antibodies used Article Title: Genetically-encoded markers for confocal visualization of single dense core vesicles Article Snippet: Materials & experimental systems n/a Involved in the study Antibodies Eukaryotic cell lines Palaeontology and archaeology Animals and other organisms Clinical data Dual use research of concern Plants Methods n/a Involved in the study ChIP-seq Flow cytometry MRI-based neuroimaging Antibodies Antibodies used Article Title: GluN2A mediates ketamine-induced rapid antidepressant-like responses. Article Snippet: Ketamine was thought to induce rapid antidepressant responses by inhibiting GluN2B-containing N-methyl-d-aspartic acid (NMDA) receptors (NMDARs), which presents a promising opportunity to develop better antidepressants.. However, adverse side effects limit the broader application of ketamine and GluN2B inhibitors are yet to be approved for clinical use.. It is unclear whether ketamine acts solely through GluN2B-dependent mechanisms. Article Title: A systematic analysis of Trypanosoma brucei chromatin factors identifies novel protein interaction networks associated with sites of transcription initiation and termination Article Snippet: Article Title: HLTF Prevents G4 Accumulation and Promotes G4-induced Fork Slowing to Maintain Genome Stability Article Snippet: The primary antibodies were diluted in 3% BSA/PBS and incubated overnight at 4°C: rabbit anti-G4 (clone 1H6, Absolute Antibody ab00389-23.0, 1:500), goat anti-G4 (clone 1H6, Absolute Antibody ab00389-24.1, 1:500), anti-G4 (clone BG4, Sigma-Aldrich MABE917, 1:500), rabbit anti-GFP (Abcam ab290, 1:1000), Article Title: Neural circuit mechanisms underlying context-specific halting in Drosophila. Article Snippet: Alexa fluor secondary antibodies ( Virus:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice Recombinant:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice Protease Inhibitor:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice Reporter Assay:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice Cloning:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice Quantitative RT-PCR:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice ChIP-qPCR:Article Title: Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice. Article Snippet: Article Linking glucose signaling to nitrogen utilization by the OsHXK7-ARE4 complex in rice |
![Figure 7. Inhibition of PKCa Activity or LSD1 Activity in Mice Shows Attenuated Septic- Induced Mortality (A) Immunoblot analysis of lungs from WT mice (n = 3 per group). Mice were intraperitoneally injected with Go6976 (1 mg/kg body weight), GSK- LSD1 (1 mg/kg body weight), or an equal volume of vehicle for 1 hr prior to LPS challenge (10 mg/kg body weight) for 6 hr. (B) WT (blue line) or Lsd1SA/SA (red line) mice were subjected to CLP (n = 20 each). Animal survival was monitored every 6 hr for 144 hr after CLP. (C and D) WT mice (n = 20) were administered with Go6976 (green line) or an equal volume of vehicle (blue line) (C) or GSK-LSD1 (purple line) or an equal volume of vehicle (blue line) (D) after CLP (1 mg/kg body weight, each, intravenous [i.v.], at 12 and 50 hr after CLP). Animal survival was monitored every 6 hr for 144 hr after CLP. **p < 0.01 (log-rank test) (B–D). (E) Representative images of H&E staining of lung sections from WT and Lsd1SA/SA mice after CLP (n = 6 per group) or from WT mice intravenously in- jected with Go6976, GSK-LSD1, or vehicle (n = 6 per group, 1 mg/kg body weight, at 12 and 50 hr after CLP). Mice were euthanized 72 hr after CLP. Scale bars, 200 mm. (F and G) Plasma concentrations of cytokines (MCP-1, IL-6, and TNF-a) were measured 72 hr after CLP in WT and Lsd1SA/SA mice (F) or in WT mice intravenously injected (1 mg/kg body weight) with Go6976 or GSK-LSD1 at 12 and 50 hr after CLP (G). (H and I) Septic injury marker (ALT, LDH, and BUN) levels were measured 72 hr after CLP in WT and Lsd1SA/SA mice (H) or in WT mice intravenously in- jected (1 mg/kg body weight) with Go6976 or GSK- LSD1 at 12 and 50 hr after CLP (I). Data are expressed as mean ± SD; n = 5; *p < 0.05, **p < 0.01, ***p < 0.001 (two-way ANOVA) (F and H). Data are expressed as mean ± SD; n = 5 per group; *p < 0.05, **p < 0.01 (unpaired two-tailed Student’s t test) (G and I). (J) Schematic model of the PKCa-LSD1-NF-kB- signaling axis showing that blocking PKCa or LSD1 activity decreases p65 protein stability and, thus, induces resistance to acute systemic inflammation, leading to increased survival rates of sepsis in Lsd1SA/SA mice compared to WT mice.](https://pub-med-unpaywalled-images-cdn.bioz.com/pub_med_ids_ending_with_5062/pm29395062/pm29395062__page12_image1.jpg)